The transurethral resection is normal, right?
Transurethral resection is a topic of concern for many friends, mastery of the prevention of scientific norms, and is especially important for protecting their own health. Today, transurethral resection of urine has become a frequent topic on many health reports. At the same time, although fatty liver liver liver liver is found in health tests, but transurethral resection of urine is normal.
If a health examination detects an elevation of transurethane, especially repeated elevation or lasting more than half a year, it is necessary for medical institutions to clarify the cause of the elevation of transurethane, and then targeted treatment. Diseases such as fatty liver, alcoholic hepatitis, drug-induced liver damage, autoimmune liver disease, can all cause elevation of transurethane. Long-term sleep deprivation or frequent sleepless nights can indeed cause slight elevation of transurethane.
It should be emphasized that the degree of transurethane elevation is not entirely consistent with the severity of liver damage, and therefore not to be taken lightly by mere slight elevation. From a broader perspective, the improvement of health awareness is driving the continuous improvement of the relevant areas. Therefore, many people believe that this is not a problem as long as a good rest rest is necessary.
Therefore, for patients with slightly elevated transurethane elevation, it is necessary to identify the cause of the disease and targeted treatment, rather than just by resting. It is widely believed that regulation and professionalisation will be the key words in the future. .
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. The fatty liver is the body's early-onset warning signal, which often indicates that the body has metabolic problems and a higher risk of subsequent diseases such as type 2 diabetes, coronary heart disease and chronic liver disease. Although the liver has not yet developed significant inflammation and fibromyalgia, if no timely intervention is made, it may in the future develop into fatty liver disease, liver fibromyalgia, and a small number of patients may even develop hepatic sclerosis or liver cancer.
Therefore, following the discovery of fatty liver, it is not possible to concentrate only on transaminases, which need to be evaluated comprehensively by taking into account indicators such as the patient's weight, waist circumference, blood sugar, uric acid, liver elasticity, and intervene as early as possible through healthy weight management, reasonable diet and scientific exercise. On the one hand, hepatitis B diagnosis cannot concentrate solely on transaminases, which must be combined with a patient's viral load, liver fibrosis status, age, absence of hepatitis or family history of liver cancer to determine whether there is a need for antiviral diagnosis. Currently, commonly used hepatitis B antiviral drugs are not yet able to kill the virus, can only inhibit long-term viral replication, stop medication, change medication, and the virus is easily rebounded.
On the other hand, the goal of chronic hepatitis B treatment is to suppress the virus for a long time. During the clinical treatment process, some patients worry about the inability to stop taking antiviral medication, or the cost. In fact, these current treatments are effective, and long-term use can reduce recurrent damage to the liver, significantly reducing the risk of hepatic sclerosis and liver cancer.
It is also relatively convenient to take one dose per day. In terms of price, most common antiviral medicines cost only a few dollars or tens of dollars a month. Long-term regulation of reasonable use of medicines can reduce recurrent damage to the liver and keep the vast majority of patients stable in the long term.
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